только для медицинских специалистов

Консультант врача

Электронная медицинская библиотека

Раздел 3 / 3
Страница 1 / 1

СПИСОК ЛИТЕРАТУРЫ

Anders H.J., Vielhauer V., Schlondorff D. Chemokines and chemokine receptors are involved in the resolution or progression of renal disease // Kidney Int. - 2003. - Vol. 63. - P. 401-415.

Burton C.J., Combe C., Walls J., Harris K.P.G. Secretion of chemokines and cytocines by tubular epithelial cells in response proteins // Nephrol. Dial. Transplant. - 1999. - Vol. 14. - P. 2628-2633.

D’Amico G. Tubulointerstitium as a predictor of progression of glomerular diseases // Nephron. - 1999. - Vol. 83. - P. 289-295.

Eddy A.A. Proteinuria and interstitial injury // Nephrol. Dial. Transplant. - 2004. - Vol. 19. - P. 277-281.

Fogo A.B. Progression versus regression of chronic kidney disease // Nephrol. Dial. Transplant. - 2006. - Vol. 21. - P. 281-284.

-oumenos D.S., Tsamandas A.C., Oldroyd S. et al. Transforming growth factor β1 and myofibroblasts: a potential pathway towards renal scarring in human glomerular disease // Nephron. - 2001. - Vol. 87(3). - P. 240-248.

-randaliano G., Gesualdo L., Ranieri E. et al. Monocyte Chemotactic Peptide 1 (MCP-1) expression in acute and chronic human nephritides: a pathogenic role in interstitial monocyte recruitment // J. Am. Soc. Nephrol. - 1996. - Vol. 7(6). - P. 906-913.

-uijarro C., Egido J. Transcription factor-κB (NF-κB) and renal disеase // Kidney Int. - 2001. - Vol. 59. - P. 415-424.

Remuzzi G., Bertani T. Is glomerulosclerosis a consequence of altered glomerular permeability to macromolecules? // Kidney Int. - 1990. - Vol. 38. - P. 384-394.

Ruiz-Ortega M., Ruperez M., Esteban V. et al. Angiotensin II: a key factor in the inflamma-

tory and fibrotic response in kidney disease // Nephrol. Dial. Transplant. - 2006. - Vol. 21. - P. 16-20.

Wolf G. Angiotensin II as a mediator of tubulointerstitial injury // Nephrol. Dial. Transplant. - 2000. - Vol. 15(6). - P. 61-63.

Для продолжения работы требуется вход / регистрация